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March 28, 2026Kidney International Reports0 citationsOpen Access

WCN26-3660 Association Between Urinary Biomarkers and Diuretic Response in Acute Heart Failure: A Cohort Analysis

DHDaisuke HiraiTSTakuryu SonouraMMMiki Matsuo

Key Result

High tonicity drives mammalian kidney maturation, allowing human collecting duct organoids to acquire vasopressin responsiveness and model congenital nephrogenic diabetes insipidus.

Key Points

  • This research aims to investigate the relationship between urinary biomarkers and diuretic response in patients with acute heart failure.
  • Conducted a cohort analysis of patients with acute heart failure.
  • Measured urinary biomarkers and their relationship to diuretic response.
  • Analyzed the effects of environmental cues on kidney maturation and function.
  • Identified specific urinary biomarkers associated with enhanced diuretic response.
  • High urinary tonicity linked to effective urine concentration and kidney maturation.
  • Mature kidney organoids showed increased responsiveness to vasopressin.

Structured PICO

P
Population
134 patients with acute heart failure (AHF)
I
Intervention
Urinary biomarkers (L-FABP, NGAL, and tAM) collected within 3 days of admission
O
Outcome
Diuretic response (DR), defined as the body weight change over the first 3 days per 40 mg of loop diuretic administered (furosemide equivalents)surrogate

Urinary biomarkers of tubular stress and congestion (L-FABP, NGAL, tAM) do not significantly correlate with diuretic response in patients hospitalized for acute heart failure.

Abstract

tonicity.This maturation process was mainly dependent on NFAT5.In scRNA-seq analysis, mouse CD organoids cultured at high tonicity in the presence of vasopressin and aldosterone overlapped with postnatal day 7 medullary CDs with high expression levels of marker genes such as Aqp2 and Slc14a2.Additionally, we have established a maturation protocol for human CD organoids.These organoids also underwent morphological and transcriptional maturation and acquired vasopressin responsiveness at high tonicity.scRNA-seq analysis revealed that the mature CD organoids exhibited comparable expression levels of maturation markers to those of adult CDs.Finally, the mature CD organoids induced from iPSCs harboring a causative AVPR2 mutation of congenital NDI lost vasopressin responsiveness.In contrast, the healthy organoids responded to vasopressin with clear AQP2 expression in the apical membrane.Conclusion: High tonicity is a driving force not only for urine concentration, but also for mammalian kidney maturation.Our mature CD organoids reached a postnatal stage, and potentially an adult stage.Our study uncovers a novel principle of kidney maturation triggered by environmental cues and offers a promising approach to maximize the potential of kidney organoids for future therapies.I have no potential conflict of interest to disclose.I did not use generative AI and AI-assisted technologies in the writing process.

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Cite This Study

Hirai et al. (2026) studied this question. High tonicity drives mammalian kidney maturation, allowing human collecting duct organoids to acquire vasopressin responsiveness and model congenital nephrogenic diabetes insipidus.

synapsesocial.com/papers/69c76fff8bbfbc51511e0683https://doi.org/10.1016/j.ekir.2026.105940
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