Why the study?
The review aimed to examine molecular cross-talk among the RAS, antioxidant pathway, inflammation, and vitamin D signaling in T2DM pathogenesis to clarify how their interactions drive disease and support mechanism-based therapies.
Design
Review
Key result
Dysregulation of the renin-angiotensin system, oxidative stress, chronic inflammation, and vitamin D signaling pathways interact to drive insulin resistance and complications in type 2 diabetes.
Authors
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Should not yet alter clinical T2DM management; extends mechanistic understanding of disease progression to inform future targeted.
The review elucidates the molecular cross-talk among RAS, oxidative stress, inflammation, and vitamin D signaling in T2DM, supporting the development of precise, mechanism-based therapeutic strategies.
Awasthi et al. (2026) conducted a review in Type 2 Diabetes Mellitus. Dysregulation of the renin-angiotensin system, oxidative stress, chronic inflammation, and vitamin D signaling pathways interact to drive insulin resistance and complications in type 2 diabetes.