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May 9, 2026Frontiers in Cardiovascular Medicine0 citationsOpen Access

A novel GDF1 frameshift mutation in a young adult with right coronary artery hypoplasia and myocardial bridging: a case report

ZXZetong XiangXWXincheng WuHCHuishuang Chen

Key Points

  • The study aims to explore the association between a GDF1 mutation and congenital heart anomalies.
  • Case report of a 24-year-old male with exertional chest pain and HCAD.
  • Coronary computed tomography angiography (CCTA) assessed coronary anomalies.
  • Whole-exome sequencing identified a GDF1 frameshift mutation.
  • Identified a novel GDF1 heterozygous frameshift mutation classified as likely pathogenic.
  • CCTA revealed right coronary artery hypoplasia with a diameter <1.5 mm.
  • The association between GDF1 mutation and congenital coronary anomalies is newly suggested.

Abstract

Background Hypoplastic Coronary Artery Disease (HCAD) is a rare congenital malformation. While linked to genes like NOTCH1, its genetic spectrum is incomplete. No prior association with GDF1 exists. Case presentation A 24-year-old male with exertional chest pain underwent coronary computed tomography angiography (CCTA), which revealed right coronary artery (RCA) hypoplasia (diameter 1. 5 mm) and a superficial myocardial bridge (SMB) of the left ramus artery. Whole-exome sequencing identified a novel heterozygous frameshift mutation in GDF1 (NM₀01492. 4: c. 84₉1del; p. Val31ArgfsTer15), validated by Sanger sequencing. According to ACMG guidelines, the variant was classified as likely pathogenic (PVS1 + PM2). Conclusion This is the first report suggesting a potential association between a GDF1 loss-of-function mutation and HCAD and SMB, expanding the phenotypic spectrum of GDF1-related disorders and providing insights into the genetic etiology of congenital coronary anomalies.

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Cite This Study

Xiang et al. (2026) studied this question.

synapsesocial.com/papers/69fece83b9154b0b82875e54https://doi.org/10.3389/fcvm.2026.1797320
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