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. Mechanistically, HERV2365 sponged miR-326, thereby derepressing fibroblast growth factor 1 (FGF1) and promoting tumor growth. Collectively, our findings identified HERV2365 as an unfavorable prognostic marker by promoting ICC progression through regulating the miR-326/FGF1 axis.
Zhong et al. (2026) studied this question.