PulseExploreJournal ClubResearchersJournals
Instagram
HomeJournal ClubExplore
Synapse
⌘+K
Synapse
June 4, 2026Journal of Biochemical and Molecular ToxicologyOpen Access

Mitigation of Cisplatin‐Induced Neurotoxicity via Nrf2/HO‐1 Signaling and Autophagy: The Protective Role of Vitamin D

View Full Paper
Ask AI
Bookmark
Share

Authors

SCSongul CuglanAYAzibe YildizKTKevser Tanbek

Discussion

Loading...

Member takes

Overview

Randomized trial examines vitamin D's effect on oxidative stress in rats, suggesting protective benefits against neurotoxicity.

Key Points

  • To investigate the effects of vitamin D treatment on cisplatin-induced oxidative stress in the brain.
  • 21 Wistar Albino rats divided into three groups: control, CP, and VitD + CP.
  • CP group received 7 mg/kg cisplatin; VitD + CP group received 1000 IU/mL vitamin D for 7 days post-treatment.
  • Biochemical and histological analyses were performed on brain tissue samples.
  • Cisplatin increased oxidative stress and pro-inflammatory cytokines, while vitamin D treatment decreased oxidative stress levels.
  • Significant increases in TOS and OSI in the CP group; however, the VitD + CP group showed significant decreases in OSI.
  • Histological examinations revealed degenerative changes in neurons of the CP group, with reduced immunoreactivity of Nrf2, HO-1, and LC3 in the VitD + CP group.

Cite This Study

Cuglan et al. (2026) studied this question.

synapsesocial.com/papers/6a2117dfd499ed480b170b12https://doi.org/10.1002/jbt.70941
View Full Paper
Ask AI
Bookmark
Share