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July 31, 2016Atherosclerosis53 citationsOpen Access

Dyslipidaemia in the Middle East: Current status and a call for action

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KAKhalid Al‐RasadiWAWael AlmahmeedKAKhalid F. AlHabib

Key Result

The Middle East exhibits a unique pattern of dyslipidaemia characterized by low HDL and high triglycerides, driven by high rates of metabolic syndrome, diabetes, and familial hypercholesterolaemia.

Structured PICO

P
Population
Populations in the Middle East region

Highlights the unique epidemiological pattern of dyslipidemia in the Middle East and calls for regional guidelines, FH registries, and preventive programs.

Abstract

The increase in the cardiovascular disease (CVD)-associated mortality rate in the Middle East (ME) is among the highest in the world. The aim of this article is to review the current prevalence of dyslipidaemia and known gaps in its management in the ME region, and to propose initiatives to address the burden of dyslipidaemia. Published literature on the epidemiology of dyslipidaemia in the ME region was presented and discussed at an expert meeting that provided the basis of this review article. The high prevalence of metabolic syndrome, diabetes, familial hypercholesterolaemia (FH) and consanguineous marriages, in the ME region, results in a pattern of dyslipidaemia (low high-density lipoprotein cholesterol and high triglycerides) that is different from many other regions of the world. Early prevention and control of dyslipidaemia is of paramount importance to reduce the risk of developing CVD. Education of the public and healthcare professionals and developing preventive programs, FH registries and regional guidelines on dyslipidaemia are the keys to dyslipidaemia management in the ME region.

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Cite This Study

Al‐Rasadi et al. (2016) conducted a review in Dyslipidaemia. The Middle East exhibits a unique pattern of dyslipidaemia characterized by low HDL and high triglycerides, driven by high rates of metabolic syndrome, diabetes, and familial hypercholesterolaemia.

synapsesocial.com/papers/6a215cbae06b4fc4c1ab9ae9https://doi.org/10.1016/j.atherosclerosis.2016.07.925
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