Key points are not available for this paper at this time.
Abstract A strain of Aedes aegypti (L.) was collected in Rio Piedras, Puerto Rico, in 1987 and reared through to the F 2 generation. The strain was shown to be heterozygous for resistance to a range of pyrethroids, but there was no evidence of organophosphate and carbamate resistance. Biochemical studies indicated that the pyrethroid resistance was not metabolically based. Electrophysiological studies at the neuromuscular junction of resistant insects indicated that a kdr (nerve insensitivity) type mechanism was operating in this strain. It is not known whether this mechanism was selected by earlier DDT exposure or by the recent ultra-low-volume applications of pyrethroids.
Hemingway et al. (1989) studied this question.