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The human body every minute tries to keep itself in homeostatic balance. The kidneys mediate this regulation by reabsorbing or excreting excess water and the ions present in it. The amount of water removed from the urine is called diuresis. The kidney has anatomy that allows the reabsorption of water in various structures, such as the distal nephron (distal tubule and collecting duct). Furthermore, above the organ, there is the adrenal gland which produces aldosterone, a hormone capable of reabsorbing Na+ in the distal tubules and reabsorbing K+. Low blood pressure secretes aldosterone, which as a result of the pressure drop activates a pathway that releases a hormone that secretes aldosterone to increase pressure, angiotensin II (ANG II). ANG II has vasoactive properties. This hormone triggers the renin-angiotensin system. ANG II functions as a signal that the adrenal cortex should release aldosterone. The process occurs as follows: (a) the juxtaglomerular cells secrete renin; (b) the function of renin will be to transform angiotensinogen (inactive) into active, angiotensin I (ANGI); (c) when the active protein is circulating in the blood, it encounters an angiotensin-converting enzyme (ACE), which it will transform into angiotensin II; (d) ANG II is released into the blood, when it reaches the adrenal gland, stimulates the synthesis and release of aldosterone and, therefore, aldosterone in the distal nephron stimulates the reabsorption of Na+ in the renal tubule.
Fuzinatto et al. (Wed,) studied this question.