MT1E binds to LncRNA NEAT1 to regulate SLC39A14-mediated ferroptosis in the pathogenesis of aortic dissection

Key Points

• Ferroptosis is significantly regulated by MT1E in relation to SLC39A14 expression, impacting aortic dissection.
• This analysis revealed the interaction between MT1E and NEAT1, which contributes to ferroptosis mechanisms.
• The approach utilized was observational analysis focusing on the molecular interactions involving both MT1E and lncRNA NEAT1.
• Understanding this pathway could enable novel therapeutic targets for aortic dissection and related diseases.