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Dioclea violacea lectin preserves brain mitochondrial function and prevents oxidative damage after ischemia/reperfusion | Synapse
March 3, 2026
Dioclea violacea lectin preserves brain mitochondrial function and prevents oxidative damage after ischemia/reperfusion
PC
Pedro Lourenzo Oliveira Cunha
MN
Maria Stella Batista de Freitas Neta
LL
Ludmila Araújo de Lima
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Key Points
Mitochondrial function is preserved after ischemia/reperfusion due to the action of Dioclea violacea lectin, enhancing brain health.
The study shows a significant reduction in oxidative damage, indicating the protective effects of lectin on neuronal cells.
Analysis employed in vitro techniques to measure mitochondrial health post-ischemia/reperfusion in brain tissues.
These findings highlight the potential for using lectin-based interventions to improve outcomes following ischemic events.
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Cunha et al. (Tue,) studied this question.
synapsesocial.com/papers/69a76079c6e9836116a2d3ea
https://doi.org/https://doi.org/10.1016/j.biochi.2026.01.017
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