Does BAF treatment during the subacute stage improve cardiac remodeling and dysfunction at the chronic stage after myocardial infarction?
Inhibition of granulation tissue cell apoptosis with BAF during the subacute stage of myocardial infarction improves chronic cardiac remodeling and dysfunction.
The postinfarct treatment with BAF improved LV remodeling and dysfunction through inhibition of granulation tissue cell apoptosis. These findings imply a new therapeutic strategy against postinfarct heart failure.
Hayakawa et al. (Tue,) studied this question.