Slow pathway ablation for dual AV nodal non-reentrant tachycardia in a 69-year-old female with tachyarrhythmia-induced cardiomyopathy significantly improved her ejection fraction and palpitations.
Case Report (n=1)
This case highlights the importance of correctly identifying dual AV nodal non-reentrant tachycardia as a reversible cause of tachyarrhythmia-induced cardiomyopathy.
We describe the case of a 69-year-old female patient who presented with a sensation of her heart pounding and an electrocardiogram showing tachycardia with unusual grouped beating. She was recently diagnosed with heart failure with reduced ejection fraction presumed to be secondary to atrial fibrillation which did not improve with beta-blocker therapy. She underwent an electrophysiology study which demonstrated Dual AV nodal non-reentrant tachycardia status post slow pathway ablation. She had significant improvement in her ejection fraction and palpitations thereafter. This case highlights the significance of correctly identifying the rhythm for management of tachyarrythmia-induced cardiomyopathy.
Cholagh et al. (Fri,) conducted a case report in Dual AV Nodal Non-Reentrant Tachycardia and Tachyarrhythmia-Induced Cardiomyopathy (n=1). Slow pathway ablation was evaluated on Improvement in ejection fraction and palpitations. Slow pathway ablation for dual AV nodal non-reentrant tachycardia in a 69-year-old female with tachyarrhythmia-induced cardiomyopathy significantly improved her ejection fraction and palpitations.