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DURING the past 10 years, investigators of radiation-induced carcinogenesis in human beings have concentrated on continuing the follow-up of populations previously studied and on collating and reanalyzing all available data. Although these approaches may not seem exciting in comparison to what has been going on in molecular biology, they are essential for two simple reasons. First, there are very few populations suitable for fruitful study, and it is unlikely that additional ones will become available. Second, it has become clear that in many types of cancer, not even 30 years of follow-up are adequate to show the total effect of . . .
Kohn et al. (Thu,) studied this question.
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