Key points are not available for this paper at this time.
axis is a central driver of redox-dependent injury, and highlight therapeutic strategies targeting this network, including zinc modulation, NO pathway regulation, peroxynitrite scavenging, and mitochondrial protection. In conclusion, targeting the Zn-NO axis as a unified redox hub may limit secondary injury propagation and improve neuroprotection in acute brain injury.
Yang et al. (Wed,) studied this question.