Our findings support a dual model of HS pathogenesis involving genetically driven epithelial dysfunction and systemic inflammation. The identification of HLA-DRA-associated variants raises the possibility that HS may share features with autoimmune diseases, while the proteomic profile dominated by innate cytokines suggests that an autoinflammatory component is also involved.
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Argyropoulou et al. (Sat,) studied this question.
synapsesocial.com/papers/69ada8b2bc08abd80d5bbea3 — DOI: https://doi.org/10.1111/jdv.70390
M. Argyropoulou
National and Kapodistrian University of Athens
Georgios Tavoulareas
National and Kapodistrian University of Athens
Joyce Leman
Kings Park Hospital
Radboud University Nijmegen
University of Bonn
Radboud University Medical Center
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