September 13, 2013Open Access

Oxidized Ca 2+ /Calmodulin-Dependent Protein Kinase II Triggers Atrial Fibrillation

APAnil PurohitAurora St. Luke's Medical Center ARAdam G. RokitaUniversity of Göttingen XGXiaoqun GuanUniversity of Iowa

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Resumen

Our studies suggest that CaMKII is a molecular signal that couples increased reactive oxygen species with AF and that therapeutic strategies to decrease oxidized CaMKII may prevent or reduce AF.

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Purohit et al. (Fri,) studied this question.

synapsesocial.com/papers/69effac7bce9831ba4f73cda https://doi.org/https://doi.org/10.1161/circulationaha.113.003313

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