Aryl hydrocarbon receptor pathway mediates cigarette smoke-induced skin inflammation and sebaceous gland hyperplasia

Key Points

• Skin inflammation and sebaceous gland hyperplasia are significantly linked to cigarette smoke exposure.
• Aryl hydrocarbon receptor activity appears heightened in response to cigarette smoke, leading to tissue changes.
• Observational analysis identifies the relationship between cigarette smoke exposure and skin pathology.
• Findings highlight the need for further investigation into interventions targeting this molecular pathway.