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SQUAMOUS-cell cancer of the cervix originates from intraepithelial precursors whose natural history has been studied extensively and is well understood.1 There is abundant evidence that these precursors are part of a single disease continuum, called cervical intraepithelial neoplasia, rather than being two separate diseases — dysplasia and carcinoma in situ.2 Several screening programs have documented a statistically significant fall in cervical-cancer incidence and death rates that can be attributed to the screening for precursors and to their eradication,3 and Boyes has demonstrated a direct correlation between the intensity of screening and the reduction of cancer in his analysis of cytology . . .
Ralph M. Richart (Thu,) studied this question.
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