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Developing a tissue-engineered model replicates blood-air barrier properties in vitro, highlighting its relevance in studying pulmonary diseases.
Background The blood-air barrier (BAB) of the lung is a critical interface responsible for gas exchange and protection against external attempts, and acts as a selective barrier. Developing in vitro models that replicate its structural and functional properties is essential in studying pulmonary diseases and their therapy.
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Özdemir et al. (2025) studied this question.
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