September 28, 2025Interventional Neuroradiology

Reconsidering dimethyl sulfoxide in endovascular embolization: A potential toxic-dependent contributor to hydrocephalus?

View Full Paper

Ask AI

Bookmark

Authors

LCLeandro Castañeyra-RuizChildren's Hospital of Orange County BHBrian W. HanakChildren's Hospital of Orange County SSSora SatoChildren's Hospital of Orange County

Discussion

Member takes

Implication

This correspondence highlights the neurotoxic potential of dimethyl sulfoxide exposure, suggesting a link to hydrocephalus development and urging for further safety evaluations.

Key Points

DMSO exposure may contribute to hydrocephalus development, indicating a need for caution in embolization techniques.
Emerging evidence suggests DMSO may cause ependymal damage and promote astrocyte apoptosis in the brain.
Neuroinflammation triggered by DMSO could lead to serious post-embolization complications in patients.
Long-term safety assessments of embolic agents should include considerations of hydrocephalus as a possible outcome.

Cite This Study

Castañeyra-Ruiz et al. (2025) studied this question.

synapsesocial.com/papers/68d90a0a41e1c178a14f65f4 https://doi.org/10.1177/15910199251380379

View Full Paper

Ask AI

Bookmark

Implication

This correspondence highlights the neurotoxic potential of dimethyl sulfoxide exposure, suggesting a link to hydrocephalus development and urging for further safety evaluations.

Reconsidering dimethyl sulfoxide in endovascular embolization: A potential toxic-dependent contributor to hydrocephalus?

Discussion

Member takes

Implication

Key Points

Cite This Study

Reconsidering dimethyl sulfoxide in endovascular embolization: A potential toxic-dependent contributor to hydrocephalus?

Discussion

Member takes

Implication

Key Points

Cite This Study

Also Consider

Also Consider