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October 1, 2024

Supplementary Figure S6 from The E3 Ubiquitin Ligase FBXO38 Maintains the Antitumor Function of Natural Killer Cells by Sustaining IL15R Signaling

Yongjing Shi Xiaodong Zheng Hui Peng Chenqi Xu Rui Sun Zhigang Tian Haoyu Sun Xianwei Wang

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Abstract

Supplementary Figure S6. FBXO38 deficiency increases the expression of suppressors of cell cycle program in TINK cells

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Shi et al. (2024) studied this question.

synapsesocial.com/papers/68e56226e2b3180350efed55 https://doi.org/10.1158/2326-6066.27142944

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Synapse has enriched 8 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Figure S5 from The E3 Ubiquitin Ligase FBXO38 Maintains the Antitumor Function of Natural Killer Cells by Sustaining IL15R Signaling2024
2Supplementary Figure S1 from The E3 Ubiquitin Ligase FBXO38 Maintains the Antitumor Function of Natural Killer Cells by Sustaining IL15R Signaling2024
3Supplementary Figure S3 from The E3 Ubiquitin Ligase FBXO38 Maintains the Antitumor Function of Natural Killer Cells by Sustaining IL15R Signaling2024
4Supplementary Figure S8 from The E3 Ubiquitin Ligase FBXO38 Maintains the Antitumor Function of Natural Killer Cells by Sustaining IL15R Signaling2024
5Supplementary Figure S4 from The E3 Ubiquitin Ligase FBXO38 Maintains the Antitumor Function of Natural Killer Cells by Sustaining IL15R Signaling2024
6Supplementary Figure S7 from The E3 Ubiquitin Ligase FBXO38 Maintains the Antitumor Function of Natural Killer Cells by Sustaining IL15R Signaling2024
7Supplementary Figure S2 from The E3 Ubiquitin Ligase FBXO38 Maintains the Antitumor Function of Natural Killer Cells by Sustaining IL15R Signaling2024
8Data from The E3 Ubiquitin Ligase FBXO38 Maintains the Antitumor Function of Natural Killer Cells by Sustaining IL15R Signaling2024