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September 20, 2024Electromagnetic Biology and MedicineOpen Access

Radiofrequency field inhibits RANKL-induced osteoclast differentiation in RAW264.7 cells via modulating the NF-κB signaling pathway

Caihua Ding Haiying Wang Chunyu Yang Hang Yang Shunxing Zhu Yi Cao

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Abstract

In this study, we investigated the inhibitory effects of radiofrequency exposure on RANKL-induced osteoclast differentiation in RAW264.7 cells, along with the underlying mechanisms. RAW264.7 cells were subjected to radiofrequency exposure at three distinct power densities: 50 µW/cm

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Cite This Study

Ding et al. (2024) studied this question.

synapsesocial.com/papers/68e57d34b6db64358751c2c2 https://doi.org/10.1080/15368378.2024.2401554

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Evaluation of culture conditions for osteoclastogenesis in RAW264.7 cellsOriginal Research · 2022 · 30 citations
2Role of RANKL in the regulation of NFATc1 and c-Src mRNA expression in osteoclast-like cellsOriginal Research · 2016 · 5 citations
3Surface RANKL of Toll-like receptor 4–stimulated human neutrophils activates osteoclastic bone resorptionOriginal Research · 2009 · 228 citations
4Lipopolysaccharide- TLR-4 Axis regulates Osteoclastogenesis independent of RANKL/RANK signalingOriginal Research · 2021 · 90 citations
5RANKL-mediated Reactive Oxygen Species Pathway That Induces Long Lasting Ca2+ Oscillations Essential for OsteoclastogenesisOriginal Research · 2010 · 208 citations

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StemRegenin 1 attenuates the RANKL-induced osteoclastogenesis via inhibiting AhR-c-src-NF-κB/p-ERK MAPK-NFATc1 signaling pathway2024

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