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September 16, 2024

Supplementary Fig S4 from Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice

Jorge Morales-Valencia Coralie Petit Alexander Calderon Siddharth Saini Gregory David

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Abstract

SIN3B deletion results in unchanged H4K16 Acetylation levels upon DSB induction.

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Morales-Valencia et al. (2024) studied this question.

synapsesocial.com/papers/68e5857ab6db643587522368 https://doi.org/10.1158/1541-7786.27035035

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Fig S4 from Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice2024
2Supplementary Fig S1 from Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice2024
3Supplementary Fig S1 from Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice2024
4Supplementary Fig S2 from Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice2024
5Supplementary Fig S2 from Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice2024

Supplementary Fig S3 from Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice2024
Supplementary Fig S3 from Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice2024
Data from Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice2024