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September 16, 2024

Supplementary Figure S6 from Oncogenic drivers and therapeutic vulnerabilities in KRAS wild-type pancreatic cancer

Harshabad Singh Rachel B. Keller Kevin S. Kapner Julien Dilly Srivatsan Raghavan Chen Yuan Elizabeth F. Cohen Michael Tolstorukov Elizabeth Andrews Lauren K. Brais Annacarolina da Silva Kimberly Perez Douglas A. Rubinson Rishi Surana Marios Giannakis Kimmie Ng Thomas E. Clancy Matthew B. Yurgelun Benjamin L. Schlechter Jeffrey W. Clark Geoffrey I. Shapiro Michael H. Rosenthal Jason L. Hornick Valentina Nardi Yvonne Li Hersh Gupta Andrew D. Cherniack Matthew Meyerson James M. Cleary Jonathan A. Nowak Brian M. Wolpin Andrew J. Aguirre

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Abstract

MAPK amplifications are alternative drivers in KRAS WT pancreatic cancer

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Singh et al. (2024) studied this question.

synapsesocial.com/papers/68e586c1b6db643587523903 https://doi.org/10.1158/1078-0432.27033430

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Figure S6 from Oncogenic drivers and therapeutic vulnerabilities in KRAS wild-type pancreatic cancer2024
2Supplementary Figure S5 from Oncogenic drivers and therapeutic vulnerabilities in KRAS wild-type pancreatic cancer2024
3Supplementary Figure S5 from Oncogenic drivers and therapeutic vulnerabilities in KRAS wild-type pancreatic cancer2024
4Supplementary Figure S7 from Oncogenic drivers and therapeutic vulnerabilities in KRAS wild-type pancreatic cancer2024
5Supplementary Figure S7 from Oncogenic drivers and therapeutic vulnerabilities in KRAS wild-type pancreatic cancer2024

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Supplementary Figure S3 from Oncogenic drivers and therapeutic vulnerabilities in KRAS wild-type pancreatic cancer2024
Supplementary Figure S3 from Oncogenic drivers and therapeutic vulnerabilities in KRAS wild-type pancreatic cancer2024
Supplementary Figure S8 from Oncogenic drivers and therapeutic vulnerabilities in KRAS wild-type pancreatic cancer2024