September 5, 2024Open Access

Innate T‐cell‐derived IL‐17A/F protects from bleomycin‐induced acute lung injury but not bleomycin or adenoviral TGF‐β1‐induced lung fibrosis in mice

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Resumen

The pathobiology of IL-17 in lung fibrogenesis is controversial. Here we examined the role of IL-17A/F in bleomycin (BLM) and adenoviral TGF-β1-induced lung fibrosis in mice. In both experimental models, WT and IL17af

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Moog et al. (Thu,) studied this question.

synapsesocial.com/papers/68e5943fb6db64358752efb7 — DOI: https://doi.org/10.1002/eji.202451323

Authors

Marie Moog

Medizinische Hochschule Hannover

Melina Baltes

Medizinische Hochschule Hannover

Tina Röpke

Medizinische Hochschule Hannover

Journals

European Journal of Immunology

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Institutions

McMaster University

Universität Hamburg

RWTH Aachen University

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Innate T‐cell‐derived IL‐17A/F protects from bleomycin‐induced acute lung injury but not bleomycin or adenoviral TGF‐β1‐induced lung fibrosis in mice

Puntos clave

Resumen

Citation Network

Connected Papers

Discussion

Cite this study

Authors

Journals

Actions

Institutions

References and Citations

Citation Network

Connected Papers

Discussion