Cardiac MRI enables quantification of EAT thickness with good to excellent reproducibility for differentiating the hypertensive patients with arrhythmias from those without arrhythmias and normal controls. Additionally, EAT thickness was correlated with native T1 and LVMi, which demonstrated EAT might induce cardiac remodeling and promote myocardial fibrosis. Cardiac MRI-derived EAT thickness metrics has great potential to further explore the mechanism of EAT with arrhythmias and guide an appropriate treatment in patients of hypertension.
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Yang et al. (2024) studied this question.
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