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July 11, 2024Archives of Physiology and Biochemistry

METTL14 plays an oncogenic role in NSCLC by modulating ferroptosis and the m6A modification of GPX4

Yang Lou Kan Huang Bo Xu Xianguo Chen

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Abstract

N6-methyladenosine (m6A) of RNA is involved in the progression of non-small cell lung cancer (NSCLC).

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Cite This Study

Lou et al. (2024) studied this question.

synapsesocial.com/papers/68e609bdb6db64358759cd4d https://doi.org/10.1080/13813455.2024.2376813

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1METTL16-dependent GPX4 m6A modification links ferroptosis to NSCLC TKIs resistance2025 · 7 citations
2Molecular mechanism of METTL7B-mediated m6A modification in ferroptosis of non-small cell lung cancer cellsOriginal Research · 2026
3METTL14-Mediated lncRNA MSTRG.292666.16 m6A Modification Promotes the Progression of Non-Small-Cell Lung Cancer Through the MAPK PathwayOriginal Research · 2026
4Methyltransferase like‐14 suppresses growth and metastasis of non‐small‐cell lung cancer by decreasing <scp>LINC02747</scp>2024 · 9 citations
5Mechanism of methyltransferase METTL14 mediating m6A modification and regulating SIRT5 expression to promote ferroptosis and repress gastric cancer progression2025

More related papers

Lactylation-Mediated METTL14 Promotes Glycolysis Through Facilitating PGAM1 m6A Methylation in Non-Small Cell Lung Cancer2026
METTL14-mediated m6A mRNA modification of G6PD promotes lung adenocarcinoma2024
The dual regulatory role of METTL14-mediated m6A modification in tumorigenesis and its underlying mechanisms2026

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