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July 2, 2024Open Access

Supplementary Figure S3 from The Novel ATR Inhibitor M1774 Induces Replication Protein Overexpression and Broad Synergy with DNA-targeted Anticancer Drugs

Ukhyun Jo Yasuhiro Arakawa Astrid Zimmermann Daiki Taniyama Makito Mizunuma Lisa M. Jenkins Tapan K. Maity Suresh Kumar Frank T. Zenke Naoko Takebe Yves Pommier

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Abstract

This figure shows protein networks in SN-38/control and correlation plots between replication fork protection proteins and VE-822.

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Cite This Study

Jo et al. (2024) studied this question.

synapsesocial.com/papers/68e61b6eb6db6435875adb32 https://doi.org/10.1158/1535-7163.26145192

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Figure S3 from The Novel ATR Inhibitor M1774 Induces Replication Protein Overexpression and Broad Synergy with DNA-targeted Anticancer Drugs2024
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3Supplementary Figure S3 from The Novel ATR Inhibitor Tuvusertib (M1774) Induces Replication Protein Overexpression and Broad Synergy with DNA-targeted Anticancer Drugs2024
4Supplementary Figure S2 from The Novel ATR Inhibitor M1774 Induces Replication Protein Overexpression and Broad Synergy with DNA-targeted Anticancer Drugs2024
5Supplementary Figure S2 from The Novel ATR Inhibitor M1774 Induces Replication Protein Overexpression and Broad Synergy with DNA-targeted Anticancer Drugs2024

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Supplementary Figure S1 from The Novel ATR Inhibitor M1774 Induces Replication Protein Overexpression and Broad Synergy with DNA-targeted Anticancer Drugs2024
Supplementary Figure S1 from The Novel ATR Inhibitor M1774 Induces Replication Protein Overexpression and Broad Synergy with DNA-targeted Anticancer Drugs2024
Supplementary Figure S2 from The Novel ATR Inhibitor Tuvusertib (M1774) Induces Replication Protein Overexpression and Broad Synergy with DNA-targeted Anticancer Drugs2024