⌘+K

Pulse Explore Communities Researchers Journals

Home Communities Explore

June 20, 2024Open Access

Cerebral cortical functional hyperconnectivity in a mouse model of spinocerebellar ataxia type 8 (SCA8)

Angela K. Nietz Laurentiu S. Popa Russell E. Carter Morgan L Gerhardt Keerthi Manikonda Laura P.W. Ranum Timothy J. Ebner

View Full Paper

Ask AI

Bookmark

Share

Abstract

Spinocerebellar Ataxia Type 8 (SCA8) is an inherited neurodegenerative disease caused by a bidirectionally expressed CTG●CAG expansion mutation in the

Discussion

No takes yet. Share an insight, caveat, or question.

Cite This Study

Nietz et al. (2024) studied this question.

synapsesocial.com/papers/68e6404bb6db6435875d1f5b https://doi.org/10.1101/2024.06.20.599947

View Full Paper

Ask AI

Bookmark

Share

Keep reading

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Cas9 Editing of ATXN1 in a Spinocerebellar Ataxia type 1 Mice and human iPSC-derived neurons2024 · 11 citations
2Hemichorea as the sole clinical manifestation of spinocerebellar ataxia type 8: a case report2025
3Molecular clues unveiling spinocerebellar ataxia type-12 pathogenesis2024 · 5 citations
4Cerebellar contribution to cognitive deficits and prefrontal cortex dysfunction in Spinocerebellar Ataxia Type 1 (SCA1)2024 · 2 citations
5T2 Hyperintensity in the Inferior Olivary and Dentate Nuclei: Novel Imaging Findings in Spinocerebellar Ataxia Type 8: A Case ReportCase Report · 2026

More related papers

Exploring functional and structural connectivity disruptions in spinocerebellar ataxia type 3: Insights from gradient analysis2024
<scp><i>STUB1</i></scp> Mutations as Possible Genetic Modifiers in Spinocerebellar Ataxia Type 82024
Spinocerebellar ataxia type 4 is caused by a GGC expansion in the <i>ZFHX3</i> gene and is associated with prominent dysautonomia and motor neuron signs2024