The present study examines the implications of acute and sub-acute exposure to cadmium chloride (CdCl2) on tissue specific glucose metabolism in muscle, gill, liver, heart, and kidney, after 30 and 60 days. Data represent significant increase in tissue glucose levels, corresponding to the metabolic response caused by cadmium-induced stress. We found out the glucose level after that 24 hour in the liver where the liver had the highest glucose levels, that the liver was going through its detox phase. Our results support a model in which there are systemic metabolic adaptations to cadmium toxicity having broader implications for how the metabolic response to exposure of an environmental toxicant is seen.
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Ramesh Chandra (2024) studied this question.
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