Early life exposure to mercury and relationships with telomere length and mitochondrial DNA content in European children

Manuel Lozano; Rosemary McEachan; John Wright; Tiffany Yang; Courtney Dow; Manik Kadawathagedara; Johanna Lepeule; Mariona Bustamante; Léa Maître; Martine Vrijheid; Anne Lise Brantsæter; Helle Margrete Meltzer; Vasiliki Bempi; Theano Roumeliotaki; Cathrine Thomsen; Tim S. Nawrot; Karin Broberg; Sabrina Llop

doi:10.1016/j.scitotenv.2024.173014

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Abstract

Telomere length (TL) and mitochondrial function expressed as mitochondrial DNA copy number (mtDNAcn) are biomarkers of aging and oxidative stress and inflammation, respectively. Methylmercury (MeHg), a common pollutant in fish, induces oxidative stress. We hypothesized that elevated oxidative stress from exposure to MeHg decreases mtDNAcn and shortens TL.

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Lozano et al. (2024) studied this question.

synapsesocial.com/papers/68e6ad90b6db64358762f48b https://doi.org/10.1016/j.scitotenv.2024.173014

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Early life exposure to mercury and relationships with telomere length and mitochondrial DNA content in European children

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Abstract

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Early life exposure to mercury and relationships with telomere length and mitochondrial DNA content in European children

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Abstract

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