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March 4, 2024Open Access

Table S2 from ERBB4-Mediated Signaling Is a Mediator of Resistance to PI3K and BTK Inhibitors in B-cell Lymphoid Neoplasms

Alberto J. Arribas Sara Napoli Luciano Cascione Laura Barnabei Giulio Sartori Eleonora Cannas Eugenio Gaudio Chiara Tarantelli Afua A. Mensah Filippo Spriano Antonella Zucchetto Francesca Maria Rossi Andrea Rinaldi Manuel Castro de Moura Sandra Jovic Roberta Bordone Pittau Anastasios Stathis Georg Stüssi Valter Gattei Jennifer R. Brown Manel Esteller Emanuele Zucca Davide Rossi Francesco Bertoni

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Abstract

Panel of antibodies used in flow cytometry experiments.

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Arribas et al. (2024) studied this question.

synapsesocial.com/papers/68e75dd0b6db6435876d4b9c https://doi.org/10.1158/1535-7163.25337702

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Table S2 from ERBB4-Mediated Signaling Is a Mediator of Resistance to PI3K and BTK Inhibitors in B-cell Lymphoid Neoplasms2024
2Table S3 from ERBB4-Mediated Signaling Is a Mediator of Resistance to PI3K and BTK Inhibitors in B-cell Lymphoid Neoplasms2024
3Table S3 from ERBB4-Mediated Signaling Is a Mediator of Resistance to PI3K and BTK Inhibitors in B-cell Lymphoid Neoplasms2024
4Table S1 from ERBB4-Mediated Signaling Is a Mediator of Resistance to PI3K and BTK Inhibitors in B-cell Lymphoid Neoplasms2024
5Table S1 from ERBB4-Mediated Signaling Is a Mediator of Resistance to PI3K and BTK Inhibitors in B-cell Lymphoid Neoplasms2024