Is the Parkinson’s-associated protein TMEM175 a proton channel: Yay or nay?

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Research reveals TMEM175 functions primarily as a K+ channel, affecting membrane flux.
TMEM175 shows minimal H+ permeability, contrary to previous beliefs about lysosomal function.
Observational analysis highlights the role of protein degradation in Parkinson's disease's pathogenesis.
Understanding TMEM175's function may clarify mechanisms underlying neurodegeneration.

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Research reveals TMEM175 is a K+ channel, indicating protein degradation may affect Parkinson’s pathogenesis.

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Freeman et al. (2025) studied this question.

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