Analysis reveals endocrine-disrupting chemicals impact erectile dysfunction through molecular pathways, suggesting targeted intervention strategies.
This study systematically characterizes the molecular pathways through which EDCs contribute to ED, with CTNNB1 and HIF1A emerging as central players. The identification of these core targets provides a theoretical foundation for developing targeted interventions against environment-related ED and underscores the importance of mitigating EDC exposure in public health strategies.
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Wang et al. (2025) studied this question.
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