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January 23, 2026Journal of Clinical Medicine7 citationsOpen Access

KRAS Inhibition in Pancreatic Ductal Adenocarcinoma

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RPRoshini PradeepNINooredeen IsbeihFAFreya F. Abraham

Key Points

  • The review aims to summarize advancements in understanding KRAS's role in pancreatic ductal adenocarcinoma and explore therapeutic strategies targeting it.
  • Reviewed historical understanding of RAS and its impact on PDAC biology.
  • Summarized development of various KRAS inhibitors, including covalent and panRAS inhibitors.
  • Explored mechanisms of resistance and feedback loops in PDAC.
  • Highlighted ongoing KRAS-specific siRNA research and immunotherapy approaches.
  • Outlined current clinical trial landscape for KRAS-targeted therapies.
  • Identified key downstream effectors and resistance mechanisms in PDAC.
  • Highlighted advancements in KRAS G12C inhibitors and other codon-specific therapies.
  • Discussed emerging strategies like targeted protein degradation and synthetic lethality.
  • Outlined progress in transitioning promising therapies from preclinical stages to early human trials.

Abstract

KRAS alterations are a hallmark of pancreatic ductal adenocarcinoma (PDAC) found in >90% of tumors. This review examines the historical evolution of the understanding of RAS and its central role in PDAC biology. We summarize the various downstream effectors, feedback loops, and resistance mechanisms that play a pivotal role in PDAC oncogenesis. Our review explores the early development of covalent inhibitors of KRAS G12C and efforts at specific inhibition of other codons and newer approaches of targeted protein degradation. We subsequently summarize the development of panRAS inhibitors and allosteric and switch-region targeting before focusing on rational therapeutic blockade of crosstalk and upstream signaling, with attention to synthetic lethality approaches transitioning from preclinical to early-phase in-human clinical trials. This review elaborates on ongoing KRAS-specific siRNA research and evolving KRAS-directed immunotherapies. We conclude by outlining the current KRAS clinical trial landscape and future areas of investigation.

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Cite This Study

Pradeep et al. (2026) studied this question.

synapsesocial.com/papers/69730f78c8125b09b0d1f4e0https://doi.org/10.3390/jcm15020873
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