PulseExploreJournal ClubDebatesTrendingResearchersJournals
Instagram
HomeExploreJournal ClubTrending
Synapse
⌘+K
Synapse
January 26, 2026Natural Product Research5 citations

Optimised Gynura divaricata polysaccharide extract ameliorated type 2 diabetes in mice by activating the PI3K/Akt/GLUT-4 signalling pathway

View Full Paper
XSXiaolong ShangJWJiapeng WangHZH. Zhang

Key Points

  • The aim is to explore the extraction and hypoglycaemic effects of polysaccharides derived from Gynura divaricata.
  • Conducted hot-water extraction followed by ethanol precipitation of Gynura divaricata polysaccharides.
  • Utilised Box-Behnken design to optimise extraction parameters like time, temperature, and ratio.
  • Isolated a homogeneous polysaccharide, GDP1, for further evaluation of its biological effects.
  • GDP1 significantly lowered fasting blood glucose levels in type 2 diabetes mellitus mice.
  • Improved insulin sensitivity and reduced inflammation and oxidative stress were observed with GDP1 treatment.
  • The extract activated the PI3K/Akt/GLUT4 signalling pathway, facilitating glucose transport.

Abstract

Type 2 diabetes mellitus (T2DM) is a long-term metabolic disorder, which is characterised by insulin resistance and persistent hyperglycaemia. Natural polysaccharides exhibit promising hypoglycaemic potential; however, optimising their extraction and mechanistic understanding of polysaccharides from Gynura divaricata (GDP) remain limited. Here, GDP underwent hot-water extraction followed by ethanol precipitation. A response surface methodology known as Box-Behnken design was utilised to refine the extraction process (extraction time, 120 min; temperature, 90 °C; solid-to-liquid ratio, 1:20 g/mL). Under these optimal conditions, the polysaccharide yield was 0.42% ± 0.03%. A homogeneous polysaccharide, GDP1, isolated and purified from GDP, exhibited potent hypoglycaemic activity in T2DM mice. GDP1 effectively decreased fasting blood glucose levels, ameliorated dyslipidemia, reduced inflammation and oxidative stress, and enhanced insulin sensitivity. GDP1 upregulated the levels of phosphorylated phosphoinositide 3-kinase (PI3K), Akt, and glucose transporter 4 (GLUT4), suggesting that its hypoglycaemic effect was partly owing to the activation of the PI3K/AkT/GLUT4 axis.

Ask AI
Helpful
Bookmark
Share
View Full Paper

Cite This Study

Shang et al. (2026) studied this question.

synapsesocial.com/papers/69770353722626c4468e84f5https://doi.org/10.1080/14786419.2026.2619418
Ask AI
Helpful
Bookmark
Share
View Full Paper