Demonstrates the effect of STAT1 on inflammation and cell behavior in rheumatoid arthritis fibroblast-like synoviocytes, suggesting therapeutic avenues.
STAT1 transcriptionally activated CXCL13 to enhance inflammation, promote proliferation, and suppress apoptosis in RA-FLS. These findings highlight the STAT1/CXCL13 axis as a potential therapeutic target for modulating synovial hyperplasia and inflammation in RA.
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Wang et al. (2026) studied this question.
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