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February 16, 2026Nano Letters0 citations

A Mutually Reinforcing Nanoplatform Orchestrates Immunogenic Ferroptosis and Reprogramming of Tumor-Associated Macrophages for Potent Immunotherapy

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LTLinlin TaoWGWeijiao GongNYNan Yang

Key Points

  • The aim is to introduce a nanoplatform that enhances immunogenicity and reprograms macrophages for improved cancer treatment.
  • Developed Hb-DD@SRF nanoplatform with sorafenib and hemoglobin components.
  • Targeted iron transporters on tumor cells to catalyze reactive oxygen species production.
  • Inhibited glutathione peroxidase 4 to amplify ferroptosis and promote immunogenic cell death.
  • Targeted M2 macrophages to counteract immunosuppression and enhance the immune response against tumors.
  • The nanoplatform significantly increased reactive oxygen species levels and induced ferroptosis.
  • Reprogrammed M2 macrophages led to enhanced immune activity in the tumor microenvironment.
  • Combined with anti-PD-L1 therapy, resulted in notable suppression of both primary and metastatic tumors.

Abstract

Solid tumors present formidable barriers to immunotherapy due to low immunogenicity and a highly suppressive microenvironment. This study introduces Hb-DD@SRF, a mutually reinforcing nanoplatform comprising a sorafenib (SRF)-loaded core coated with a dual-targeting hemoglobin (Hb) shell. Upon targeting iron transporters on tumor cells, Fe2+ derived from Hb catalyzes reactive oxygen species (ROS) generation, while acidic conditions trigger SRF release to inhibit glutathione peroxidase 4 (GPX4), synergistically amplifying ferroptosis. This robust process elicits immunogenic cell death to prime T cells. Crucially, Hb-mediated oxygenation precisely offsets the severe hypoxia resulting from SRF, establishing a complementary loop that prevents drug resistance. Furthermore, the platform targets M2 macrophages via the haptoglobin pathway, where oxygen and SRF jointly reprogram them to reverse immunosuppression. This remodeled immunostimulatory microenvironment synergizes with anti-PD-L1 therapy to achieve pronounced suppression of primary and metastatic tumors. Collectively, Hb-DD@SRF orchestrates ferroptosis amplification and comprehensive microenvironment modulation to potentiate antitumor immunotherapy.

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Cite This Study

Tao et al. (2026) studied this question.

synapsesocial.com/papers/69926503eb1f82dc367a0d47https://doi.org/10.1021/acs.nanolett.5c06063
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