Findings demonstrate that anti-VEGF therapies improve glial-vascular interactions in a rat model of retinopathy of prematurity, suggesting therapeutic potential.
These findings suggest that pathological angiogenesis in ROP is mediated through VEGF- and mTORC1-dependent mechanisms. Anti-VEGF therapies may help restore glial-vascular interactions by reducing abnormal blood vessels in the ROP retina.
No takes yet. Share an insight, caveat, or question.
Deguchi et al. (2026) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: