Research suggests descending monoaminergic pathways influence motility in colitis, indicating their role in gut inflammation.
This study suggested that persistent noxious input from an inflamed colon activates descending monoaminergic neurons, leading to enhanced basal motility in noninflamed regions. Our findings provide important insights into the pathophysiology of inflammation-associated dysmotility.
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Yuki et al. (2026) studied this question.
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