Demonstrates protective effects against retinal damage in vivo and in vitro, highlighting potential therapeutic uses.
AME exerts protective effects against BL-induced retinal degeneration and oxidative stress-induced photoreceptor apoptosis, potentially through activation of the Nrf2 pathway. These findings highlight the therapeutic potential of AME in preventing or mitigating retinal damage associated with oxidative stress.
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Wu et al. (2026) studied this question.
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