Demonstrates MSC-sEV reduce Th2 airway inflammation in mice, suggesting a potential therapy for lung conditions.
MSC-sEV are able to alter the metabolic state of DCs, favoring DCs to maintain OXPHOS (oxidative phosphorylation) rather than glycolysis, thereby reducing DCs-initiated inflammatory responses and attenuating Th2 lung inflammation, suggesting MSC-sEV can be a potential clinical therapy for airway inflammation.
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Huang et al. (2026) studied this question.
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