Empowering exhausted T cells of Glioblastoma patients by Neurotransmitters and Neuropeptides: decreasing immune checkpoint inhibitors, and increasing CD3zeta, proliferation and Glioblastoma arrest

Mia Levite; Nili Ilouz; Eithan Galun; Yigal Shoshan

doi:10.1007/s00262-025-04226-6

January 27, 2026SHILAP Revista de lepidopterologíaOpen Access

Empowering exhausted T cells of Glioblastoma patients by Neurotransmitters and Neuropeptides: decreasing immune checkpoint inhibitors, and increasing CD3zeta, proliferation and Glioblastoma arrest

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Key Points

Increased proliferation and CD3zeta among T cells was observed, emphasizing the role of neurotransmitters and neuropeptides.
The study highlights a significant decrease in immune checkpoint inhibitors, potentially enhancing T cell activity.
Observational analysis examined T cells' responses in glioblastoma patients with varying neurotransmitter and neuropeptide levels.
Findings indicate a need for further investigation into neuropeptide effects on tumor arrest and T cell function.

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Implication

Observational analysis reveals decreased immune checkpoint inhibitors and increased CD3zeta in glioblastoma patients, suggesting potential therapeutic implications.

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Cite This Study

Levite et al. (2026) studied this question.

synapsesocial.com/papers/69a75b16c6e9836116a21c15 https://doi.org/10.1007/s00262-025-04226-6

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Empowering exhausted T cells of Glioblastoma patients by Neurotransmitters and Neuropeptides: decreasing immune checkpoint inhibitors, and increasing CD3zeta, proliferation and Glioblastoma arrest

Key Points

No abstract yet

Implication

Discussion

Cite This Study

Empowering exhausted T cells of Glioblastoma patients by Neurotransmitters and Neuropeptides: decreasing immune checkpoint inhibitors, and increasing CD3zeta, proliferation and Glioblastoma arrest

Key Points

No abstract yet

Implication

Discussion

Cite This Study

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