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March 3, 2026International Journal of Molecular Sciences3 citationsOpen Access

Interactions Between Nutraceuticals and α-Synuclein Conformational States: Molecular Mechanisms and Neuroprotective Implications in Parkinson’s Disease

BABruna AmentaRMRosalba MinerviniMMMaria Laura Matrella

Key Points

  • Nutraceuticals can modulate alpha-synuclein aggregation by interfering with its structure and assembly, promoting neuroprotection.
  • Polyphenols and peptides may suppress toxic oligomer formation, which is crucial in neurodegenerative processes.
  • This review summarizes molecular mechanisms impacting alpha-synuclein's conformational states and supports clinical applications.
  • Challenges in bioavailability and blood-brain barrier penetration limit clinical translation of these nutraceuticals.

Abstract

Synucleinopathies, including Parkinson's disease (PD), are neurodegenerative disorders characterized by aberrant aggregation of α-synuclein (α-syn), a presynaptic protein with an intrinsic disorder nature. The transition of soluble monomers into oligomeric and fibrillar species represents a key molecular event driving neuronal dysfunction and neurodegeneration. Emerging evidence suggests that nutraceuticals, bioactive compounds derived from dietary sources, can modulate α-syn aggregation at multiple conformational stages. Polyphenols, alkaloids, ginsenosides, and food-derived peptides interfere with α-syn structure and assembly, suppressing the formation of toxic oligomer species and promoting the clearance of misfolded assemblies. Despite this potential, clinical translational of nutraceuticals is currently limited by poor systemic bioavailability and restricted central nervous system penetration due to blood-brain barrier constraints, which have largely confined research to preclinical studies. In this context, this review summarizes current knowledge of nutraceutical interventions targeting the conformational landscape of α-syn and highlighting both direct and indirect molecular mechanisms with involved in aggregation-prone species. Furthermore, we critically examine key challenges related to bioavailability and clinical translation, focusing on advanced delivery systems and precision-based approaches to enhance neuroprotective efficacy and support the potential of nutraceuticals as novel or adjunctive therapeutic strategies for PD.

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Cite This Study

Amenta et al. (2026) studied this question.

synapsesocial.com/papers/69a75ca7c6e9836116a25b52https://doi.org/10.3390/ijms27031324
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