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Research shows TGF-β1 disrupts prostaglandin E2 signaling in endometrial tissue, suggesting new fertility therapies.
Endometrial primary cilia are indispensable for decidualization and are potential biomarkers for predicting endometrial receptivity. Targeting TGF-β signaling to restore ciliated cell number and ciliary length may serve as a potential therapeutic strategy to improve fertility outcomes.
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Hou et al. (2026) studied this question.
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