Reply to the Complex Etiology of Sertraline‐Induced Lipid Storage Myopathy and Acquired Multiple Acyl‐ CoA Dehydrogenase Deficiency ( MADD )‐like Syndromes: Hidden Genetic Variation, Nutritional Deficiency, and Mitochondrial Vulnerability

Q: What does this research mean for the field?

Sertraline can induce lipid storage myopathy and acquired multiple acyl-CoA dehydrogenase deficiency-like syndromes due to hidden genetic variations, nutritional deficiencies, and mitochondrial vulnerabilities. Novelty: ClaimNovelty.NOVEL_FINDING. Consensus alignment: ConsensusAlignment.CHALLENGES_CONSENSUS.

Aziz Shaibani; Alexis Taylor

doi:10.1002/mus.70214

March 12, 2026Muscle & Nerve

Reply to the Complex Etiology of Sertraline‐Induced Lipid Storage Myopathy and Acquired Multiple Acyl‐ CoA Dehydrogenase Deficiency ( MADD )‐like Syndromes: Hidden Genetic Variation, Nutritional Deficiency, and Mitochondrial Vulnerability

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Key Points

The aim is to explore the multifactorial causes behind sertraline-induced lipid storage myopathy and MADD-like syndromes.
Review of existing literature
Identification of hidden genetic variations
Analysis of nutritional deficiencies and mitochondrial vulnerabilities.
No datasets were generated or analyzed as part of this article.

Implication

Analysis reveals genetic and nutritional factors in lipid storage myopathy and MADD-like syndromes.

Abstract

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Cite This Study

Shaibani et al. (2026) studied this question.

synapsesocial.com/papers/69b64d48b42794e3e660e0d2 https://doi.org/10.1002/mus.70214

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Reply to the Complex Etiology of Sertraline‐Induced Lipid Storage Myopathy and Acquired Multiple Acyl‐ CoA Dehydrogenase Deficiency ( MADD )‐like Syndromes: Hidden Genetic Variation, Nutritional Deficiency, and Mitochondrial Vulnerability

Key Points

Implication

Abstract

Discussion

Cite This Study

Reply to the Complex Etiology of Sertraline‐Induced Lipid Storage Myopathy and Acquired Multiple Acyl‐ CoA Dehydrogenase Deficiency ( MADD )‐like Syndromes: Hidden Genetic Variation, Nutritional Deficiency, and Mitochondrial Vulnerability

Key Points

Implication

Abstract

Discussion

Cite This Study

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