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March 18, 2026Heart and Vessels2 citationsOpen Access

The impact of mildly elevated HbA1c level, and low levels of high-density lipoprotein cholesterol and hemoglobin on microvascular angina

SSSatoru SuzukiKYKenshi YamanagaMIMasanobu Ishii

Key Result

Mildly elevated HbA1c (OR 1.96 per 1%), low HDL-C (OR 0.59 per 10 mg/dL), and low hemoglobin (OR 0.52 per 1 g/dL) were independently associated with microvascular angina.

Key Points

  • This research aims to understand clinical factors contributing to microvascular angina (MVA).
  • Conducted a retrospective case-control comparison involving 92 MVA patients and 391 controls.
  • Utilized coronary angiography, ACh-provocation tests, and CFR measurements for diagnosis.
  • Employed multivariate logistic regression to analyze factors linked to MVA.
  • Mildly elevated HbA1c, low HDL-C, and low hemoglobin independently associated with MVA.
  • Lower mean corpuscular volume found in MVA patients compared to controls.
  • No differences in clinical characteristics between MVS and MVA without MVS.

Study Design

Type

Case-Control (n=483)

Multicenter

No

Structured PICO

P
Population
483 participants, including 92 patients with microvascular angina (MVA) diagnosed by COVADIS criteria (underwent coronary angiography with ACh-provocation and CFR measurement) and 391 age- and sex-matched controls without chest pain or cardiovascular diseases. Mean age 63.9 years, 58% women, based in Japan.
O
Outcome
Clinical factors associated with microvascular angina (MVA)

Mildly elevated HbA1c, low HDL-C, and low hemoglobin are independently associated with microvascular angina, suggesting they may serve as modifiable risk markers.

Main Result

Effect estimate: OR 1.96 (95% CI 1.38-2.79)

p-value: p=<0.001

Limitations

  • Single-center study with a relatively small sample size
  • Retrospective study design
  • Results may not be applicable to patients outside the studied age range
  • Lack of information on medications taken by control participants
  • Potential for asymptomatic CAD in control participants
  • FlowWire could theoretically influence coronary artery tone
  • CFR was not measured in all study patients and control participants

Abstract

Microvascular angina (MVA), resulting from coronary microvascular dysfunction (CMD), is increasingly recognized as a contributor to adverse cardiovascular outcomes. However, the clinical factors for MVA remain unclear. In this retrospective case-control study, we investigated the clinical factors of MVA in 92 patients with MVA and 391 age- and sex-matched control participants without chest pain or cardiovascular diseases. All patients underwent coronary angiography including an acetylcholine (ACh)-provocation test and a coronary flow reserve (CFR) measurement. MVA was diagnosed based on the Coronary Vasomotion Disorders International Study Group (COVADIS) criteria. Multivariate logistic regression analysis showed mildly elevated hemoglobin A1c (HbA1c) (i.e., prediabetes), low high-density lipoprotein cholesterol (HDL-C), and low hemoglobin (Hgb) levels as independent factors significantly associated with MVA. Mean corpuscular volume (MCV) was also significantly lower in MVA patients. No significant differences in clinical characteristics were found between microvascular spasm (MVS) and MVA without MVS. MVS was more common in women than in men. In conclusion, mildly elevated HbA1c level, and low levels of HDL-C and Hgb may serve as modifiable risk markers for MVA. The study findings may aid in the management of MVA.

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Cite This Study

Suzuki et al. (2026) conducted a case-control in Microvascular angina (n=483). Mildly elevated HbA1c, low HDL-C, and low hemoglobin vs. Control participants without chest pain or cardiovascular disease was evaluated on Independent clinical factors associated with microvascular angina (OR 1.96, 95% CI 1.38-2.79, p=<0.001). Mildly elevated HbA1c (OR 1.96 per 1%), low HDL-C (OR 0.59 per 10 mg/dL), and low hemoglobin (OR 0.52 per 1 g/dL) were independently associated with microvascular angina.

synapsesocial.com/papers/69ba43984e9516ffd37a4fc2https://doi.org/10.1007/s00380-026-02660-9
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