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March 21, 2026Neuropharmacology0 citationsOpen Access

Aggression after intermittent ethanol intoxication in mice: Sex differences and modulation via medial amygdala relaxin-3/RXFP3 signaling

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MZMohamed Aly ZahranAMAroa Mañas-OjedaMNMónica Navarro-Sánchez

Key Points

  • This research investigates the effects of intermittent ethanol exposure on aggressive behaviors in mice, highlighting sex differences and related neurobiological pathways.
  • Examined dominance and aggression through tube-dominance and resident-intruder tests in mice after intermittent ethanol intoxication.
  • Analyzed neural activation patterns using c-Fos protein expression in brain regions associated with aggression.
  • Administered RXFP3 agonist via AAV injection into the medial amygdala of male mice to assess behavioral outcomes.
  • Male mice showed increased dominance and aggression during acute abstinence from ethanol.
  • Female mice exhibited heightened defensive behaviors rather than aggression after ethanol exposure.
  • Relaxin-3 immunoreactivity in the medial amygdala was linked to recovery in aggressive behaviors in males.
  • Chronic RXFP3 agonist treatment in the medial amygdala significantly reduced aggressive behaviors in male mice.

Abstract

Alcohol consumption is strongly associated with aggression and violence in humans, yet the underlying neurobiological mechanisms within key aggression circuits remain poorly understood. In this preclinical study, we examined the effects of intermittent alcohol intoxication on dominance and aggressive behaviors in mice, focusing on sex differences and the potential involvement of the nucleus incertus relaxin-3/relaxin-family peptide receptor 3 (RXFP3) signaling pathway. Using an intermittent ethanol-intoxication protocol, we observed that male mice displayed a transient increase in dominance and aggressive behaviors during acute abstinence, as measured by the tube-dominance and resident-intruder tests, whereas female mice displayed heightened defensive responses. Distinct patterns of neural activation across brain regions, reflected by c-Fos protein expression, were associated with aggression in males, including decreased expression in the medial amygdala (MeA) and increased expression in the ventromedial hypothalamus (VMH), consistent with an established MeA-VMH based aggression circuit. Additionally, the levels of relaxin-3 immunoreactivity in MeA nerve fibers increased in parallel with behavioral recovery, suggesting a modulatory role of relaxin-3/RXFP3 signaling. To test this hypothesis, we bilaterally injected an adeno-associated viral (AAV) vector expressing the selective RXFP3 agonist, R3/I5, into the MeA of male mice. Notably, this chronic localized R3/I5 treatment significantly reduced dominance and aggressive behaviors both before and after alcohol intoxication. Together, these data demonstrate that relaxin-3/RXFP3 signaling in the MeA counteracts alcohol-related aggression in male mice, pointing to this pathway as a potential target for treating impulsive violence associated with alcohol intoxication in humans. • Intermittent ethanol intoxication increases dominance and aggression in male mice. • Neural activity in aggression circuits alters after acute alcohol abstinence. • Female mice display defensive, not aggressive responses after ethanol intoxication. • Relaxin-3/RXFP3 signaling in medial amygdala alters alcohol-related aggression. • RXFP3 agonist in medial amygdala reduces dominance/aggression in male mice.

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Cite This Study

Zahran et al. (2026) studied this question.

synapsesocial.com/papers/69be35166e48c4981c6732f3https://doi.org/10.1016/j.neuropharm.2026.110924
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