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March 21, 2026Nutrients2 citationsOpen Access

Broccoli-Derived Exosome-like Nanoparticles Alleviates Metabolic Dysfunction-Associated Steatotic Liver Disease Through Modulating the Gut–Liver Axis

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FZFeng ZhangRLRuolan LiuTXTongxiao Xu

Key Points

  • This research aims to explore the therapeutic effects of broccoli-derived exosome-like nanoparticles on metabolic dysfunction-associated steatotic liver disease.
  • Fractionated broccoli-derived exosome-like nanoparticles using differential centrifugation.
  • Randomized 24 male C57BL/6J mice into control, high-fat diet (HFD), and BDENs groups.
  • Administered 100 mg/kg of BDENs to the BDENs group for 4 weeks after HFD modeling.
  • Analyzed liver injury markers, intestinal barriers, cecal content metabolomics, and serum inflammatory factors.
  • Identified 1659 microRNAs linked to immunity and fatty acid biosynthesis.
  • Significantly reduced weight and ALT/AST ratio in BDENs group (p < 0.05).
  • Attenuated liver histopathological damage and lipid accumulation.
  • Maintained intestinal barrier and regulated bile acid metabolism.
  • Reduced serum LPS level (p < 0.01) and suppressed markers of hepatic inflammation (p < 0.0001).

Abstract

Background/Objectives: Metabolic dysfunction-associated steatohepatitis (MASLD) represents a prevalent liver disease worldwide. It is crucial to maintain the stability of the gut–liver axis in order to inhibit the advancement of MASLD. Broccoli-derived exosome-like nanoparticles (BDENs) can alleviate constipation and improve colitis. This study investigated whether BDENs possess therapeutic potential for improving induced MASLD by the gut–liver axis. Methods: BDENs were fractionated from fresh broccoli using differential centrifugation, and the microRNAs were identified and analyzed. 24 male C57BL/6J mice (6 weeks old) were randomized into the control group, HFD group, and BDENs group, with 8 mice per group. After 8 weeks of high-fat diet modeling, the BDENs group accepted BDENs daily oral gavage of 100 mg/kg (B.W.), while the control and HFD groups accepted 1 × PBS. Four weeks after BDENs intervention, analysis was conducted on liver injury markers, liver tissue pathology, intestinal barrier, cecal content metabolomics and fecal 16S rRNA, serum inflammatory factors, and hepatic inflammation. Results: BDENs identified 1659 miRNAs associated with physiological processes such as immunity, antioxidant defense, and fatty acid biosynthesis. BDENs significantly reduced weight and ALT/AST ratio (p < 0.05). Furthermore, BDENs attenuated hepatic histopathological damage and lipid accumulation. For the gut–liver axis, BDENs maintained intestinal barrier, regulated intestinal bile acid metabolism and restored the gut microbiota. Additionally, BDENs reduced serum LPS level (p < 0.01) and suppressed hepatic inflammation, including F4/80 and IL-6, IL-1β (p < 0.0001). Conclusions: Oral BDENs therapy demonstrates potential for ameliorating MASLD.

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Cite This Study

Zhang et al. (2026) studied this question.

synapsesocial.com/papers/69be35946e48c4981c673f5fhttps://doi.org/10.3390/nu18060953
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