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Demonstrates targeted ferroptosis improves RPE function in MERTK-deficient retinitis pigmentosa, suggesting a new treatment approach.
Ferroptosis is a critical driver of MERTK-deficient RP, influencing RPE dysfunction through the MERTK/NFE2L2/HMOX1 axis. These insights highlight the potential therapeutic strategy of targeting ferroptosis to regulate RPE function in the treatment of MERTK-deficient RP.
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Wen et al. (2026) studied this question.
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